LEI 11418 PDF

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Gene Ontology GO Terms. The relative expression of osteogenic and bone resorption markers was determined by real-time polymerase chain reaction and Western blotting, respectively.

Read Article at publisher’s site. Find all citations in this journal default. We investigated the effects of GBE on glucose consumption, glucokinase activity, and mRNA levels of key genes in glucose metabolism and the insulin signaling pathway. Abstract Our aim is to investigate the potential therapeutic value of morin against osteoporosis and elucidate the mechanism of action. Either your web browser doesn’t support Javascript or it is currently turned off.

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Abstract Ginkgo biloba, an herbal medication, is capable of lowering glucose, fat, and lipid peroxide in diabetic patients. No matching affiliation detected. In the current study, we tested the hypothesis that Ginkgo biloba extract GBE prevented hyperinsulinism-induced glucose intolerance in hepatocytes.

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Here, we uncovered the therapeutic effect of morin against osteoporosis and demonstrated its suppressive action on the MAPK pathway in this disease. Journal of Natural Medicines [24 Jul72 4: Body weight was regularly monitored. Gene Ontology GO Terms. How does Europe PMC derive its citations network?

Journal of Natural Medicines [03 Sep65 1: Europe PMC requires Javascript to function effectively. Llei data indicated that GBE repressed glucose uptake under normal conditions, while it dramatically improved glucose tolerance under insulin-resistant conditions.

Body mineral density BMD was determined at proximal femurs using dual energy X-ray absorptiometry.

Furthermore, after analyzing gene expression, we suggest that GBE chiefly exerts its effects by stimulating IRS-2 transcription. It should be noted that, unlike rosiglitazone, GBE did not stimulate excessive glucose uptake as it improved glucose tolerance. It is said that GBE treatment could avoid drug-induced obesity. In the latter case, please turn on Javascript support in your web browser and reload this page.

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Morin treatment also increased the number of trabecular bones in DEX-induced rats. Mechanistically, morin reversed the decrease of osteogenic markers and increase of bone resorption markers, which might eventually be mediated by modulation of MAPK signaling cascades. To better show its efficacy, we included a control group that was treated with rosiglitazone, a type of thiazolidinedione 11481. Body weight and BMD were both significantly decreased in osteoporotic rats, although BMD was partially restored by intraperitoneal morin administration.

Our data suggest that GBE has the potential to prevent insulin resistance and is a promising anti-diabetic drug.

Europe PMC requires Javascript to function effectively. Pathological examination was performed by hematoxylin and eosin staining. CitePeer Related Articles http: